Health News

Thursday, April 3, 2014 - 16:00

Inter-species transmission of prions is fortunately a generally inefficient process, although devastating when it occurs. An exception to this is the case of a rodent called the bank vole which is universally susceptible to prion transmission from multiple other species. This tendency was exploited in a paper from researchers in the University of California to help gain information on prevention of prion transmission. The paper is published on 3rd April in the journal PLoS Pathogens.


Thursday, April 3, 2014 - 14:40

Combating the tissue degrading enzymes that cause lasting damage following a heart attack is tricky. Each patient responds to a heart attack differently and damage can vary from one part of the heart muscle to another, but existing treatments can’t be fine-tuned to deal with this variation.


Thursday, April 3, 2014 - 12:35

Researchers at the University of California, San Diego School of Medicine have found that too little or too much of an enzyme called SRPK1 promotes cancer by disrupting a regulatory event critical for many fundamental cellular processes, including proliferation.


Thursday, April 3, 2014 - 12:28

New research suggests that drugs commonly used to prevent organ rejection after transplantation may also be helpful for combating HIV. The findings, which are published in the American Journal of Transplantation, suggest a new strategy in the fight against HIV and AIDS.


Thursday, April 3, 2014 - 12:02

Stephen Wiley, a lifelong cowboy from Terrell, has helped UT Southwestern Medical Center pioneer a new treatment for vocal cord cancer. Mr. Wiley volunteered to be the world’s first known patient to be treated for vocal cord cancer with Cyberknife, a surgeon-controlled robotic device that destroys tumors with highly precise doses of radiation.


Thursday, April 3, 2014 - 07:25

A team of researchers from five Swedish universities, led by Karolinska Institutet and the Science for Life Laboratory, have identified a new way of treating cancer. The concept is presented in the journal  Nature  and is based on inhibiting a specific enzyme called MTH1, which cancer cells, unlike normal cells, require for survival. Without this enzyme, oxidized nucleotides are incorporated into DNA, resulting in lethal DNA double-strand breaks in cancer cells.